早老综合症所致ALT肿瘤的特有p53突变-p53S与Ras协同的分子基础
序号 | 标题 | 类型 | 作者 |
---|---|---|---|
1 | [The cooperation between p53 and Ras in tumorigenesis]. | 期刊论文 | Wei, Yong-Yong|Hou, Jing|Tang, Wen-Ru|Luo, Ying| |
2 | 癌蛋白YAP1的研究进展 | 期刊论文 | 郭海强|刘同阳|贾舒婷|罗瑛| |
3 | Two mechanisms underlying the loss of p16(Ink4a) function are associated with distinct tumorigenic consequences for WS MEFs escaping from senescence | 期刊论文 | Zhang, Xiufeng|Si, Xiaoyu|Tang, Wenru|Luo, Ying| |
4 | The Gain of Function of p53 Mutant p53S in Promoting Tumorigenesis by Cross-talking with H-RasV12 | 期刊论文 | Jia, Shuting|Zhao, Lanjun|Tang, Wenru|Luo, Ying| |
5 | Current Advances of Ras Induced Senescence and The Bypass Mechanism | 期刊论文 | Guo Yu-Sheng|Zhang Ru-Yi|Jia Shu-Ting|Luo Ying| |
6 | Twist1在肿瘤发生中的作用 | 期刊论文 | 蒋红果|邵杨|罗瑛| |
7 | p53 codon 72 polymorphism and breast cancer risk: A meta-analysis | 期刊论文 | Hou, Jing|Jiang, Yuan|Tang, Wenru|Jia, Shuting| |
8 | Loss of p16(Ink4a) Function Rescues Cellular Senescence Induced by Telomere Dysfunction | 期刊论文 | Zhang, Xiufeng|Wu, Xiaoming|Tang, Wenru|Luo, Ying| |